A 4-day-old newborn was delivered to the clinic. From the history of the disease it is known: a child from a second pregnancy, full-term, birth weight 3500g. The pregnancy was normal and the delivery was normal. The child began to cry immediately. When trying to give the child sweet water after the first greedy sips, a cough, cyanosis and vomiting appeared. When feeding from a spoon with expressed breast milk, vomiting, cyanosis immediately appeared, and foam flowed through the nose. Vomit without signs of curdling. On the third day, the doctor suspected a developmental defect in the child. What pathology did the doctor suspect if, on X-ray, the stomach and intestines are filled with air, and the contrast agent does not enter them? What are the mechanisms behind this embryopathy?
Esophageal atresia is a result of cell death disorder. The tracheoesophageal fistula persisted due to impaired cell differentiation and proliferation during the formation of the esophagus and trochea. Food enters the respiratory system, does not enter the stomach, as evidenced by the lack of contrast in it.
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